Expert advice and information

Read articles prepared by our thyroid experts - Prof Akheel Syed, Prof Marian Ludgate, Prof Peter Taylor, Dr Angelos Kyriacou and Prof Kristien Boelaert

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How does the thyroid affect my weight?

Hyperthyroidism and weight changes - Dr Angelos Kyriacou, Consultant in Endocrinology & Diabetes, Limassol, Cyprus

Thyroid hormones affect all the tissues and organs in the body and are important for maintaining our ‘internal balance’. They are very important for maintaining our ‘energy balance’ i.e. how much energy we consume (via eating) and how we use and store energy in our tissues and organs (a process referred to as ‘metabolism’). In simple terms, thyroid hormones affect our appetite and how we ‘burn’ this food to be healthy and function well in our day-to-day activities.

Hyperthyroidism (overactive thyroid) refers to increased thyroid hormone production from the thyroid gland. Hyperthyroidism and its treatment are often associated with large fluctuations in weight. Weight changes observed with hyperthyroidism and its treatment can vary quite a lot from person-to-person as well; whilst some people are unaffected, others are significantly affected, and this is something we will discuss in this article.

I have hyperthyroidism. How will that affect my weight?

Weight loss is a common symptom in people with hyperthyroidism. Up to 90% of people diagnosed with hyperthyroidism will report weight loss. However, the remaining 10% will either report that their weight does not change or, occasionally, that it increases. So, why is there such a variability? It is thought that the increased thyroid hormones in the blood increase the appetite but increase the metabolism even more. This leads to weight loss in the majority of patients. However, where the increase in a patients’ appetite is greater than the increase in their metabolism, this can lead to weight gain.

The amount of weight loss that accompanies hyperthyroidism is often quoted at 11-15 pounds (5-7 kilograms), or 16% of the total pre-illness weight. Nevertheless, as mentioned there is great variability from person-to-person. Other factors can make a difference. These include geographical location and timing of presentation from onset of symptoms among others.

I am being treated for hyperthyroidism. What will happen to my weight?

In most people, weight will increase as the hyperthyroidism is corrected and thyroid levels are brought under control. Traditionally, any weight gain seen after diagnosis and treatment was thought to be similar to the disease-related weight loss, thus restoring weight to pre-illness levels. However, there is some emerging evidence of possible weight overshoot, i.e., weight gain that is more than the disease-related weight loss. Studies that described a weight overshoot estimated this to be anything between 2-35 pounds (1-16 kilograms). Some recent studies have shown that the frequency of obesity in patients who were previously treated for hyperthyroidism is about 50% higher than that of the general public; men and women were respectively 70% and 30% more likely to be obese, compared with the general public.

Who is at risk of excessive weight gain?

This is the subject of ongoing scientific research. Nevertheless, based on existing research we can say that the strongest risk factors for excessive weight gain are:

  • Hypothyroidism: The treatments given for hyperthyroidism (e.g. radioactive iodine and surgery) often induce hypothyroidism, with levothyroxine therapy needed. Patients whose thyroid function is not well controlled may be more at risk of excessive weight gain.
  • Graves’ disease: People with the autoimmune variety of hyperthyroidism.
  • Disease-related weight loss: If there is large disease-related weight loss then this appears to be associated with larger weight gains after treatment.
  • Radioactive iodine therapy: This is a good long-term treatment option for hyperthyroidism. However, it possibly results in mildly more weight gain than other treatments. On the other hand, some recent evidence obtained by combing data from different studies, suggests that thyroid surgery can also cause significant weight gain, which is comparable to that of radioactive iodine therapy.
  • Severity of hyperthyroidism initially: It appears that the more severe the disease before treatment, the greater the weight gain after treatment.

Other possible risk factors include sex (with men possibly being more likely to gain weight), ethnicity and smoking status. Although stopping smoking is associated with modest weight gains, it is highly recommended in hyperthyroidism because of the strong link between smoking and thyroid eye disease.

What can I do to control my weight?

There is unfortunately a scarcity of evidence to answer this question specifically for people with hyperthyroidism. Having said that, we do not have any reason to believe that the principles of weight management differ in these groups of patients, compared with the general public. Therefore, if you are concerned about excessive weight gain, there is no need to panic! There are things you can do to control your weight. The following steps, listed in order of importance, can all help with this:

  • Lifestyle changes. Firstly, ensure that you have a balanced diet, which includes lots of fruit and vegetables. This means eating a wide variety of foods in the right proportions. The key is to reduce the daily caloric intake. A clinical dietitian may guide you how to do this. You may also consider reducing your alcohol intake because alcoholic drinks are often high in calories and cause weight gain. We also encourage exercise e.g. 150 minutes of moderate physical activity, such as walking for 30 minutes at least five days per week.
  • Weight loss medications. Drugs such as Liraglutide and Semaglutide may be available on the NHS for patients who are obese. Eligibility is based on Body Mass Index (BMI - which is calculated based on patients’ weight and height), medical history and comorbidities. Eligibility criteria are the same for people with and without hyperthyroidism, for example a BMI>35 kg/m2 without other comorbidities or BMI of 30-35 kg/m2 in the presence of other comorbidities, such as Type 2 diabetes.*
  • Before starting on weight loss medication there needs to be careful consideration and discussion between patients, the endocrinologist and the obesity medicine specialist about the suitability for such medications and the timing of their use.

A low-calorie diet and exercising should be tried before embarking on weight loss medications. Conversely, even when weight loss medications are used, adopting a good lifestyle with dieting and exercise appears to be associated with better weight loss as opposed to simply taking the medications.

In summary, hyperthyroidism is usually associated with weight loss. Following its treatment, there may be excessive weight gain in some patients. The good news is that there are steps you can take to limit the amount of weight you may gain following treatment. Please look at the BTF’s weight loss and wellbeing resources for further information.

*Please note that NHS criteria with regard to BMI can vary between England and Scotland so it is important to ask your GP about the criteria that apply in your area.

What is the scientific reason that starvation diets don’t work?

Starvation diets - Prof Akheel Syed, Consultant physician in Obesity Medicine, Diabetes & Endocrinology at Salford Royal NHS Foundation Trust

Lifestyle and dietary management are the cornerstones of any healthy weight management programme. Whilst most popular diets rely on calorie restriction for weight loss, some may take this to extremes.

What is a starvation diet?

Whilst there is no medically acceptable definition of a starvation diet, the Encyclopædia Britannica describes it as a ‘a diet that does not provide a person with enough food to be healthy.’ A starvation diet may mean different things including ‘water diet’ and ‘fasting diet.’ Intermittent low energy diets, such as the 5:2 diet, are not regarded as starvations diets. A medically supervised, very low-calorie diet (VLCD, less than 800 Calories/day) is regarded as a semi-starvation diet.

If you wish to follow an intermittent low calorie diet it is advisable to seek medical advice before you start it. It may be necessary to supplement with minerals and/or vitamins to ensure you are getting the minimum daily requirements.

Can you lose weight with a starvation diet?

Yes, drastic calorie restriction can lead to rapid weight loss. Much of the initial weight loss comes from using up of glycogen (a form of storage carbohydrate) in the liver and muscles and associated water losses. This manner of weight loss, however, is neither healthy nor sustainable.

What are the harms of starvation dieting?

The human body has just a few days’ worth of glycogen stores. Once glycogen stores are used up, blood sugar levels fall and the body shifts its reliance on energy to fat breakdown resulting in ketosis, causing tiredness, confusion, mood swings, fatigue and weakness of muscles. This has a negative impact on health and wellbeing and reduces physical activity.

Prolonged starvation (beyond a week) leads to muscle atrophy (loss of muscle bulk and quality). As muscle mass is a crucial component of the body’s basal metabolic rate, loss of muscle slows down the metabolic rate to conserve calories. Several defensive mechanisms come into play to protect the body from excessive weight loss, slowing down weight loss and breaking the will to fast, resulting in failure of the diet with bingeing on ’prohibited’ food choices. This is not a failure of ‘willpower’ but a strong biological urge to protect the body from the dangers of starvation. This can then lead to feelings of frustration, guilt, shame, reduced self-worth, and wanting to regain control. This starts a new yo-yo diet cycle with the predictable results shown in the illustration.

Other harms of starvation diets include deficiencies of micronutrients and vitamins such as iron, folate and thiamine which can have serious effects. Resuming food intake after a period of starvation dieting also carries the risk of ‘refeeding syndrome’ — a potentially serious condition resulting from large changes in fluids and salts, phosphate, glucose and vitamin levels in the blood. Thus, starvation diets are very hazardous, even more so to people with underlying health issues such as diabetes or heart disease.

Where can I get help with managing my weight?

The NHS has many resources for a healthy lifestyle and a balanced diet such as Live Well, Better Health, and a smartphone app, NHS Weight Loss Plan (available on the Apple App Store or Google Play Store). You can also speak to your GP or practice nurse for available resources local to you and, if clinically appropriate, about referral to specialist weight management services.

What is an elimination diet?

Can elimination diets help with weight loss? Prof Akheel Syed, Consultant physician in Obesity Medicine, Diabetes & Endocrinology at Salford Royal NHS Foundation Trust

"Elimination diets" have soared in popularity in recent years. But are they good for weight loss?

An elimination diet is a trial to identify foods or ingredients to which you may have an intolerance or allergy. Some well-known trigger foods include milk/dairy, egg, peanut, tree nuts, soy, wheat, fish and shellfish.

Why do an elimination diet?

If you think you have a food allergy, make an appointment with your GP and discuss it with them. For example, if you suspect you have a gluten intolerance it is important to get this confirmed/diagnosed. People with an autoimmune thyroid disease have a greater risk of having coeliac disease than someone without.

Symptoms of a food allergy can include:

  • tingling or itching in the mouth
  • an itchy red rash (hives or urticaria)
  • hay fever-like symptoms, such as sneezing or itchy eyes
  • nausea (feeling sick) or vomiting
  • abdominal pain or diarrhoea

Your doctor may advise blood and skin tests to identify some food allergies. You may need them before you can safely try an elimination diet.

Elimination diets are not advised if you have symptoms of severe allergic reactions such angioedema (swelling of the face, mouth, or throat) or anaphylaxis:

  • feeling lightheaded or faint
  • breathing difficulties – such as fast, shallow breathing
  • wheezing or shortness of breath
  • a fast heartbeat
  • clammy skin
  • confusion and anxiety
  • collapsing or losing consciousness
How to do an elimination diet

An elimination diet comprises two parts - an elimination (avoidance) phase, and a reintroduction (challenge) phase.

Elimination (avoidance) phase

The first step is to stop consuming particular foods or ingredients. You’ll need to check food labels and ask how food is prepared when eating out. And remember to consider food additives. For example, if you decide to eliminate onions and garlic, you should be aware that these are often found as additives in pre-prepared products or foods like crisps. You should aim to keep a food diary and write down everything you eat and note how you feel after you have eaten it.

Types of elimination diets

There are several types of elimination diets. Your doctor or dietitian can design one that’s right for you. Some common types are:

  • Simple modified diet: this basic elimination diet involves avoiding just one or two foods. If you suspect you feel unwell after eating dairy products for example, you might just start with avoiding those.
  • Moderate intensity diet: several groups of food are eliminated simultaneously.
  • Strict, few foods diet: only a selected group of foods is permitted. As this is not a nutritious diet, it is not advisable for lengthy periods.
  • A low FODMAP (Fermentable Oligosaccharides, Disaccharides, Monosaccharides and Polyols) diet is a special type of elimination diet for people with conditions such as irritable bowel syndrome (IBS). A low FODMPA diet should only be tried once all other dietary advice regarding IBS has been tried first and under the guidance of a FODMAP trained professional.

It is important to eat other foods that provide the same nutrients as the food you need to avoid. When avoiding dairy products, for example, other foods fortified with calcium are advisable. You may need to consider over-the-counter supplementation of a multivitamin. Alternatively, a dietitian can help you design an elimination diet with replacement nutrients. If you regularly take prescribed medications you should check with a pharmacist whether the diet is compatible with them.

Reintroduction (challenge) phase

After eliminating possible food allergy triggers, foods are reintroduced into the diet one at a time. This helps to identify which foods trigger symptoms.

If you experience any symptoms of a severe allergy, get emergency medical help immediately and stop the elimination diet until your doctor says it is safe to resume.

The elimination (avoidance) and reintroduction (challenge) cycles are repeated until the trigger foods/ingredients are identified as shown in the illustration.

Once the foods that cause symptoms are identified, you can avoid them whilst enjoying other foods as normal.

Can elimination diets help with weight loss?

Some weight loss plans advocate elimination diets that exclude specific food groups such as dairy or wheat or a whole range of foods, usually without a medical reason.

Restrictive diets rarely help with meaningful weight loss. Sticking to calorie restriction is particularly difficult when the diet is limited in important food groups. Elimination diets often replace eliminated foods with highly processed high-calorie products, leading to weight gain rather than weight loss.

They are also frequently more expensive than normal foods. Wheat-free or gluten-free products are twice the cost, for example. There can be unpleasant side effects such as constipation (in low-carb diets, for example) or diarrhoea (due to replacement insoluble fibre) and deficiencies of micronutrients and vitamins. Each food group provides important and essential nutritional benefits as part of a healthy balanced diet and therefore if they are excluded there could be serious side effects.

The inability to lose weight can lead to feelings of frustration, guilt, shame, reduced self-worth, and yo-yo dieting as discussed in the article about Starvation diets.

Where can I get help with managing my weight?

The NHS has many resources for a healthy lifestyle and a balanced diet such as Live Well, Better Health, and a smartphone app, NHS Weight Loss Plan (available on the Apple App Store or Google Play Store). You can also speak to your GP or practice nurse for available resources local to you and, if clinically appropriate, about referral to specialist weight management services.

Genetics of thyroid disease

Do the genetics of thyroid disease explain the problems thyroid patients have?, Prof Peter Taylor, consultant endocrinologist, University Hospital of Wales

Twin studies have shown an individual’s thyroid hormone levels are largely genetically determined. Large studies with detailed genetic data have substantially increased our knowledge of the genetic architecture of TSH and, to a lesser extent, free T4 levels. It is also well established that in health the set point of an individual’s thyroid hormone levels vary much less within an individual than the range across the general population. Taken together this indicates that when thyroid hormone levels become abnormal due to a thyroid disorder, even when they are brought back to within the normal population reference-range using antithyroid drugs, such as carbimazole, or with levothyroxine, this may be outside their own genetic setpoint.

The consequences of this for an individual are unclear, but it is reasonable to think that this might have consequences for their weight over the long-term. This is because we know that even modest changes in thyroid status within the population reference-range are associated with changes in a variety of clinical outcomes including body-composition.

Most of what we know about the genetic determinants of thyroid status have related to thyroid function levels within the blood. However, the impact of thyroid hormone levels within the cells of the body is harder to identify. Rare genetic variants in thyroid hormone transporters and nuclear receptors can have profound effects on thyroid hormone levels within different parts of the body without there being substantial effects on thyroid hormone levels in the blood. It is therefore possible that more common genetic variants with much more modest effects may cause certain individuals to be more affected with more modest changes in thyroid status. However, the evidence of their impact is limited. A common variant in DIO2 (Thr92Ala) is of particular interest, but more research is needed.

It is likely that some individuals will have more trouble with their weight as a result of thyroid dysfunction due to their genetic make-up, the way their body uses and takes up thyroid hormones and their genetic risk factors for obesity. Our ability to manage these individuals differently, and to identify those individuals most at risk, remains limited. More research is urgently needed in this area.

What is my metabolic setpoint ? Is my metabolism fixed?

An individual’s basal metabolic rate is the amount of energy per unit of time that they need to keep their body functioning at rest. This accounts for around 60-70% of daily calorie expenditure. Typical processes involved in the basal metabolic rate including breathing, blood circulation, muscle and temperature regulation. Age is a key factor in an individual’s metabolic rate, but so is thyroid status.

When an individual has an overactive thyroid (hyperthyroidism) their basal metabolic rate rises and this explains some of the weight loss they may experience. In contrast, in patients with an underactive thyroid (hypothyroidism) the basal metabolic rate falls leading to weight gain. Correction of thyroid dysfunction will therefore affect an individual’s metabolic rate. Correction of hyperthyroidism is associated with weight gain, and it is important to avoid over-treatment and subsequent hypothyroidism to reduce unwanted weight gain.

References

You are what you eat

Thyroid hormones, body composition and weight loss - Prof Marian Ludgate, Professor Emerita Molecular Endocrinology, Cardiff University School of Medicine.

We are all aware of the expression ‘you are what you eat’ – but what exactly is our body composed of?

The main components are water, fat, bone and muscle. Body composition measures the proportions of these in an individual, using a weak electrical current, which will pass more quickly through water and muscle than through fat or bone. This method was applied to people with thyroid problems; those with an overactive thyroid had less muscle and bone whereas those with an underactive thyroid had increased fat and water. These changes often translate into weight loss in the former and weight gain in the latter groups of people.

When we think of weight our thoughts turn to fat. We use the Body Mass Index (BMI) as a measure of percentage body fat. However, BMI underestimates fat percentage in obese people and overestimates it in the slender, in whom muscle contributes more to weight than fat tissue. Some consider the waist-to-hip ratio as a more accurate indicator and many sophisticated weighing scales can provide information on several metrics which can be relayed to a smart device worn on the wrist.

Key facts about fat

We need fat! It stores excess energy to keep us going when food is unavailable; it keeps us warm, provides a cushion to protect vital organs and secretes hormones and factors which regulate our appetite and help insulin to act efficiently. Unfortunately too much fat, especially in the wrong place, can lead to the secretion of substances that increase inflammation and prevent insulin action. This can lead to type 2 diabetes.

Not all fat is the same! The properties I describe above are mostly found in white fat located under the skin and in our abdomens. Brown fat does not store excess calories but is able to turn energy from food into heat. It has mostly been studied in rodents and young children and was thought to be almost absent in adult humans. Its main function is to maintain body temperature in cold conditions (of obvious benefit in newborns and infants). More recent imaging methods have demonstrated that brown fat deposits exist around the neck, along the backbone and in the chest space. Of interest, the amount of brown fat detected in people was found to be inversely correlated with BMI, i.e. slim people had more brown fat and vice versa.

Apart from low temperature, brown fat is also regulated by thyroid hormones and changes in its activity may contribute to some of the symptoms associated with thyroid dysfunction.

Scientists in Switzerland measured how well temperature was maintained following cold exposure in people with underactive thyroid before and after treatment with replacement thyroid hormone (levothyroxine). They found a marked improvement in the ability to keep warm in the cold once thyroid hormone levels were restored to normal.

In contrast, German scientists used imaging to evaluate brown fat in people with an overactive thyroid before and after anti-thyroid drug (ATD) treatment. Brown fat activity was higher but areas of white fat and skeletal muscle were lower before ATD when FT4 levels were high.

These studies provide one explanation for the heat and cold intolerance experienced by those with over- or underactive thyroids respectively but can brown fat levels shed any light on weight gain?

A third type of fat exists! This is known as Beige (predictably) or Brite (for brown in white). This fat starts as white fat but can be induced to mimic brown fat and turn energy into heat. Since white fat stores excess energy but brown fat burns it as heat, browning of fat has been suggested as a way of treating obesity.

Apart from sitting in a cold office, several factors have been shown to increase brown fat/Brite fat. These include smoking (which is not recommended given its wide-ranging negative impact on health, including thyroid eye disease) and various plant-derived products such as capsaicin (found in chilli peppers) and resveratrol (a component of red grapes). Antibiotics seem to have an adverse effect on browning of fat suggesting that the bacteria and other micro-organisms in the gut (known as the gut microbiome) may be implicated in the process. The role of the gut microbiome in thyroid disease, and how it is influenced by diet, stress and other life factors, will be covered in a separate article.

Does exercise lead to fat browning ? In mice and rats the answer is definitely yes; when rodents run on a wheel, cell markers of Brite tissue are increased in fat located under the skin (sub-cutaneous) but not in the fat around the abdominal organs (visceral). In humans, the results are less convincing, but this may be due to the focus on sub-cutaneous fat which is easier to sample. Further studies are needed, including visceral fat, to determine whether fat browning can be added to the list of the many benefits associated with exercise.

How body composition changes in people with thyroid disorders?

So why does body composition change in people with thyroid issues? Thyroid hormones enter the cell’s nucleus and affect expression of genes involved in the regulation of bone turnover; too much thyroid hormone leads to an imbalance between bone formation and bone resorption which leads to weaker bones. The opposite occurs with too little thyroid hormone; although it is unclear whether people with an underactive thyroid have stronger bones. Similarly thyroid hormones regulate the cellular machinery that generates fat (also known as lipid) from excess calories and breaks it down to release energy when needed. People with an underactive thyroid have increased lipid levels, including cholesterol, in their circulation; the opposite occurs when the thyroid is overactive but in both cases these return to normal with treatment.

To assess thyroid function several different hormones can be measured; thyroxine (T4) and triiodothyronine (T3) are made by the thyroid but the activity of the gland is controlled by Thyroid Stimulating Hormone (TSH).

TSH is made by the pituitary and acts on the thyroid on/off switch, the TSH receptor. Where the thyroid is working correctly, the level of TSH keeps the amounts of T4 and T3 in balance. If the thyroid gland fails to make enough T4 and T3, and TSH levels increase to try to compensate. The opposite happens when excess T4 and T3 are produced, i.e. TSH production is reduced.

Most overactive thyroid cases are caused by an autoimmune disease in which antibodies, called Thyroid Stimulating Antibodies (TSAB) behave like TSH. Consequently, the TSH receptor is activated in almost all instances of thyroid dysfunction but, depending on the type of disease, is accompanied by too little or too much T4 and T3 The thyroid switch (TSHR) is found in many cell types outside the thyroid, including stem cells. These ‘simple’ but potent cells have the ability to change, or differentiate, into more specialised cells such as fat, bone and muscle. The differentiation processes occur throughout life, initially as part of normal growth and then to repair damaged tissues in adults.

Our experiments suggest that when TSAB activate the switch in people with an overactive thyroid, this increases browning of fat, and this usually corresponds to the period of weight loss. Anti-thyroid treatment brings T4 and T3 levels into the normal range but the TSAB take much longer to clear and may even persist. In that situation, the browning of fat reduces, less heat is produced and this may contribute to the weight gain experienced by many once normal thyroid function is restored.

Hopefully adjustments to lifestyle, i.e. healthy eating and increased exercise, can help to counteract the unhelpful impacts of thyroid disease on body composition.

References

  • Bioelectrical impedance assessment of body composition in thyroid disease. Seppel, T Kosel, A Schlaghecke, R European Journal of Endocrinology 1997 136:493-498 DOI10.1530/eje.0.1360493
  • Cypess AM, Lehman S, Williams G, Tal I, Rodman D, Goldfine AB, Kuo FC, Palmer EL, Tseng Y-H, Doria A, Kolodny GM, Kahn CR (2009) Identification and importance of brown adipose tissue in adult humans. N Engl J Med 360:1509–1517.
  • Resolution of Hypothyroidism Restores Cold-Induced Thermogenesis in Humans Maushart, CI, Loeliger, R, Gashi, G, Christ-Crain, M, Betz, MJ Thyroid 29:493-501 DOI10.1089/thy.2018.0436
  • Effects of Hyperthyroidism on Adipose Tissue Activity and Distribution in Adults Steinhoff, KG, Krause, K, Linder, N , Rullmann, M, Volke, L ,; Gebhardt, C , Busse, H, Stumvoll, M Bluher, M Sabri, O, Hesse, S , Tonjes, A Thyroid 2021 31:519-527 DOI10.1089/thy.2019.0806
  • Brown fat and obesity: the next big thing? Mark Stephens, Marian Ludgate and D. Aled Rees Clinical Endocrinology 2011 74:661–670 doi: 10.1111/j.1365-2265.2011.04018.x
  • Understanding the Biology of Thermogenic Fat: Is Browning A New Approach to the Treatment of Obesity? Ariana Vargas-Castillo, Rebeca Fuentes-Romero, Leonardo A Rodriguez-Lopez, Nimbe Torres, Armando R Tovar Arch Med Res 2017 J48:401-413. doi: 10.1016/j.arcmed.2017.10.002
  • Townsend, L.K., Wright, D.C. Looking on the “brite” side exercise-induced browning of white adipose tissue. Pflugers Arch - Eur J Physiol 471, 455–465 (2019).
  • The molecular action of thyroid hormones on bone. D Bassett and G Williams. Trends in Endocrinology and Metabolism 2003 14:356-364 DOI10.1016/S1043-2760(03)00144-90
  • The Influence of Thyroid Pathology on Osteoporosis and Fracture Risk: A Review. Dragos Apostu , Ondine Lucaciu , Daniel Oltean-Dan , Alexandru-Dorin Mures, an, Cristina Moisescu-Pop, Andrei Maxim and Horea Benea Diagnostics 2020, 10, 149; doi:10.3390/diagnostics10030149
  • Systemic regulation of adipose metabolism Christopher M.Carmean, Ronald N.Cohen, Matthew J.Brady. Biochme Biophys Acta 2014 1842:424-430
  • TSH receptor activation and body composition. de Lloyd, A; Bursell, J; Gregory, JW; Rees, DA; Ludgate, M J Endocrinology 2010 204:13-20 DOI10.1677/JOE-09-0262
  • The role of Thyrotropin receptor activation in adipogenesis and Modulation of Fat Phenotype Draman, MS; Stechman, M; Scott-Coombes, D ; Dayan, CM; Rees, DA; Ludgate, M ; Zhang, L Frontiers Endocrinology 2017 Vol 8 article 83 DOI10.3389/fendo.2017.00083

Can gut bugs affect thyroid disease?

Diet, stress and gut bugs – relevant to thyroid disease? Prof Marian Ludgate, Professor Emerita, Cardiff University School of Medicine

We know that thyroid disease ‘runs in families’ which suggests that our genes have some part to play in who gets a problem with their thyroid. Many studies have identified small variations in genes involved in controlling the immune system, as being important risk factors. However, it is possible to have identical twins where one develops thyroid disease but the other does not. This suggests that the environment in which we live, including what we eat, must also contribute to disease.

In recent years a lot of attention has been paid to the bugs living in our guts. Once thought of as something to avoid (remember to wash your hands!) we now know that the bugs help with digesting our food and much more besides. They produce a variety of soluble factors that can affect a range of processes, including the immune system.

The gut microbiota is made up of bacteria, viruses and fungi and can be analysed via their microbiome (DNA from the micro-organisms). Most cases of over- and underactive thyroid are caused by autoimmunity, i.e. when the body’s immune system mistakes components of the thyroid gland as ‘alien’ and generates antibodies that can stimulate the thyroid or T cells which lead to its destruction. The gut microbiota has been found to affect immune balance with beneficial micro-organisms increasing the ‘regulatory’ and unhelpful microorganisms promoting the ‘inflammatory’ arms of the immune system. In mice, a particular type of bacterium, known as SFB for short, was shown to increase the numbers of unhelpful T cells which promote inflammation and autoimmune conditions. There is no direct equivalent of SFB in humans but we found that amounts of their closest relative were increased in people with an overactive thyroid who did not respond well to anti-thyroid drug treatment.

We do not yet know enough about the gut microbiome to advise which bacterial types should be encouraged or discouraged or how this might be achieved. In general, a favourable microbiome has many different types of bacteria and is found in people who eat a healthy diet, especially one rich in fibre. It is no surprise that people in the west tend to have a less healthy microbiome, with low diversity, especially those who eat lots of sugary and processed foods.

We inherit our microbiome from our mothers during birth. Babies born by Caesarean section have a differing gut microbiome from those delivered via the birth canal and differences are also seen when comparing breast-fed and bottle-fed infants. The gut microbiome evolves with the introduction of solid foods, during adolescence in response to hormone changes and then remains fairly stable until old age when it becomes less diverse.

What other factors affect the microbiome? Using antibiotics alters the microorganisms present but fortunately, the effects are short-lived, except when antibiotics have to be used over a long period. Whether antibiotic treatment to remove unhelpful bacteria might provide new treatment approaches remains to be tested. The opposite – introducing ‘good bugs’ – has proven useful in people infected with antibiotic-resistant bacteria.

Smoking changes the microorganisms in the gut and in the nose and throat but quitting smoking allows the gut microbiome to recover. Stress can also take its toll; it has long been noted that people with an overactive thyroid may have experienced a stressful event in the months prior to diagnosis. Hence it is not surprising that stress, probably via the stress hormone cortisol, reduces the different types of bacteria present, including one of the types that we identified as being less plentiful in people with Graves’ disease.

How can we encourage a healthy microbiome? Prebiotics and probiotics

Prebiotics can be thought of as food for bacteria, in the sense that they contain nutrient material that we are unable to digest but our gut bugs can. Their main component is complex carbohydrate and can be found in pulses such as peas and beans, whole grains and vegetables – all items recommended as being part of a healthy diet.

Probiotics contain specific living microorganisms and are found in natural yoghurt and many fermented foods such as sauerkraut and kimchi. Probiotic formulations are also available in the form of tablets or liquids. Tablets may have coatings to increase the chances of the bacteria surviving the acid in the stomach and then reaching the correct part of the gut.

As I mentioned above, we do not yet know enough about the gut bugs which might be helpful or harmful in people with over- or underactive thyroid problems. To date there have been no large controlled clinical trials of probiotic effects in thyroid disease, although some are planned. We undertook a small study in people with Graves’ disease and found minor benefits in some patients but the work has not yet been published. Hence we cannot yet suggest a particular probiotic formulation although natural food sources containing live microorganisms are fine. As discussed in week 3’s article on diets and supplements, a healthy diet is recommended, particularly one that is rich in the complex carbohydrates (or prebiotics) that our gut bugs love best.

Conclusions

Apart from the general health benefits of supplements such as vitamin D during the winter, there is very little evidence to support taking supplements to improve thyroid problems or response to treatment. Carnitine may help if you have irregular heartbeat due to an overactive thyroid, selenium is of benefit to people with mild thyroid eye disease.

Excluding gluten is helpful only in people with both thyroid and coeliac diseases; similarly avoiding lactose is of benefit only in people with both thyroid disease and lactose intolerance.

It is wise to discuss any planned diet changes with your doctor adding a supplement or eliminating a food group. For further information about ‘Elimination diets’ please see Professor Akheel Syed’s article about this.

To encourage healthy gut bugs, eat plenty of fruit, vegetables, whole grains and pulses but avoid processed foods. Having said that the occasional ‘ready meal’ will do no harm, especially if it avoids stress at the end of a long day!

References

Supplements and exclusions

Diet supplements, exclusions and thyroid disease – Prof Marian Ludgate, Professor Emerita, Cardiff University School of Medicine

People with an over- or underactive thyroid often seek ways to improve their lifestyle with the aim of enhancing their response to medication, reducing their symptoms and returning to ‘normal’. The changes might involve quitting smoking (always a good idea), increasing the amount and variety of exercise undertaken and/or altering the quality and quantity of food, including dietary supplements, taken in.

It is estimated that almost a third of people with an underactive thyroid alter their diet by taking supplements or avoiding certain food groups. Websites and patient support groups are full of suggestions and accounts of how a specific change has been of benefit to an individual. No one doubts the importance of ‘feeling in control’ that lifestyle changes can induce, but deciding which option might work for you can be overwhelming and lead to stress.

Rather than relying on these personal testimonies, the best evidence comes from controlled clinical trials of the type used to test and validate new treatments and compare them to existing therapies. This article will focus on some of the dietary supplements and food exclusions that have been suggested to improve outcomes in people with thyroid problems – mainly the very few that have been tested in well-controlled trials. Week 3 of this programme includes an article with excellent information on several supplements/food exclusions, including iodine, soya, vitamin D, selenium, calcium and ginger so will not be discussed here.

Is there a role for supplements?

The use of food supplements has greatly increased in recent years and is a multimillion pound industry. As mentioned in week 3, supplements can be difficult to dose due to low levels of purity, meaning that the concentration given on the label may be inaccurate. A second problem with supplements is the lack of large well-controlled trials to test whether they are of real benefit.

Carnitine is one of the supplements for which there is reasonable information when applied to overactive thyroid conditions but not underactive. Carnitine is found naturally in all tissues and body fluids. During the normal process of releasing energy from food, a type of activated oxygen is released which can damage our cells. Carnitine helps to reduce this effect. Our bodies make about one quarter of the carnitine we need, the rest is provided by our diet and is found in red meat. The first studies were conducted in athletes who were shown to benefit from carnitine supplements, especially long-distance runners. A trial of carnitine in people with an overactive thyroid found that it improved many of the symptoms of hyperthyroidism, particularly irregular heartbeat.

Resveratrol acts in a similar way to carnitine, it is found in red wine but unfortunately there are no proper trials of its usefulness in the thyroid context.

Lemon balm or Melissa Officinalis, also has the same mode of action as carnitine and resveratrol; all are antioxidants. I was unable to find any trial of lemon balm in people with thyroid disorders but a review of trials of its use in other health settings found that it could improve sleep, skin quality and the circulation. The information suggesting that lemon balm may be beneficial in Graves’ disease was obtained using laboratory experiments from the 1980s. These showed that components of lemon balm could prevent the thyroid stimulating antibodies which cause Graves’ disease from binding to the thyroid on/off switch.

A more recent report described two individual women whose overactive thyroids were improved by lemon balm. Their stimulating antibody levels decreased but this could have happened spontaneously as the disease follows its natural course. These laboratory experiments and case reports suggest that lemon balm may help Graves’ disease patients but controlled clinical trials are required before it can be recommended.

What about excluding food groups?

Among the many food exclusions reported to benefit thyroid disease, gluten is one of the favourites. People with coeliac disease must follow a gluten-free diet. Like most thyroid diseases, it is an autoimmune condition affecting about 1% of the population, mainly women. The two conditions can occur together with up to 20% of people with coeliac disease also having an underactive thyroid in the 1990s, although this proportion has reduced in recent years to about 5%.

A small Polish study investigated whether a gluten-free diet would benefit women with a form of underactive thyroid together with antibodies suggesting coeliac disease. After 6 months, women on the gluten-free diet had lower thyroid autoantibodies and increased vitamin D levels than women who continued to eat gluten, but there was no difference in standard thyroid function tests. The authors suggested that improved absorption of vitamin D helped in reducing the thyroid antibodies. Other studies suggest that a gluten-free diet improves absorption of levothyroxine medication.

It has been suggested that avoiding dairy products may be of help in thyroid disease. Lactose intolerance is another autoimmune condition that can occur in people with thyroid autoimmunity. Lactose intolerance is quite widespread, being more frequent in some countries and with most people displaying no symptoms. It is more common in those with an underactive thyroid; people who have both conditions may benefit from restricting milk products and a small trial in Turkey reported better thyroid function tests on a lactose-free diet, again probably due to improved absorption of levothyroxine.

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